Investigation about proteins brochure
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Scientists at the University of Leeds discovered that a small change or misfold in the beta-2 microglobulin protein can make it unstable and prone to sticking to other proteins, starting the process that leads to amyloid fibres forming in diseases like Alzheimer's and Parkinson's. A separate study found that protein-protein recognition between an enzyme and unfolded peptide occurs through hydrophobic interaction with two specific areas of the unfolded peptide, clarifying how this important recognition process works. Advances in understanding protein folding and interactions are important for developing treatments for diseases caused by amyloid proteins and furthering research into physiological processes.
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1) Scientists at the University of Leeds discovered that a small change or misfold in the beta-2 microglobulin protein makes it unstable and prone to aggregation, starting the process of amyloid protein formation in diseases like Alzheimer's and diabetes.
2) Researchers led by Professor Thomas Kiefhaber found that protein recognition between the unstructured S-peptide and S-protein occurs through hydrophobic interaction with specific areas of the unfolded S-peptide, clarifying the protein recognition process.
3) Advances in understanding protein interactions and structures are important for developing treatments for diseases that involve amyloid proteins or are affected by changes in protein structure.
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The document summarizes three research seminars attended by the author. The first seminar presented new techniques for analyzing crystals, including thermo gravimetric analysis, differential thermal analysis, and mass spectrometry. The second seminar discussed research on how nerve growth factor in rats is modified under conditions of high blood pressure. The third seminar presented new treatment approaches for epilepsy based on studying transcription factors to generate pluripotent cells that can replace damaged cells.
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The document summarizes two studies about proteins that cause mutations. One study found that APOBEC proteins, which are involved in immune activities, cause over 70% of mutations in cancers like cervical, bladder, and lung tumors. The mutations may be influenced by environmental factors. The second study was the first to perform full genome sequencing on people with autism and found genetic risk factors for autism that could lead to improved treatments and allow for earlier diagnosis through genetic screening.
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Plegable de biologia molecular.
This document discusses oxidative stress and its relationship to diseases like diabetes and cancer. It proposes studying whether oxidative radicals that attack red blood cells cause variations in electrical potential. The study of cell membranes and the interactions between lipids and proteins is also discussed, noting their importance for cell structure and function. Methods like immunofluorescence, x-ray fluorescence, and crystallography are proposed to better understand these components and interactions at a molecular level.
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Plegable biologia molecular
1) Scientists at the University of Leeds discovered that a small change or misfold in the beta-2 microglobulin protein makes it unstable and prone to aggregation, starting the process of amyloid protein formation in diseases like Alzheimer's and diabetes.
2) Researchers led by Professor Thomas Kiefhaber found that protein recognition between the unstructured S-peptide and S-protein occurs through hydrophobic interaction with specific areas of the unfolded S-peptide, clarifying the protein recognition process.
3) Advances in understanding protein interactions and structures are important for developing treatments for diseases that involve amyloid proteins or are affected by changes in protein structure.
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The document summarizes three research seminars attended by the author. The first seminar presented new techniques for analyzing crystals, including thermo gravimetric analysis, differential thermal analysis, and mass spectrometry. The second seminar discussed research on how nerve growth factor in rats is modified under conditions of high blood pressure. The third seminar presented new treatment approaches for epilepsy based on studying transcription factors to generate pluripotent cells that can replace damaged cells.
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The document summarizes two studies about proteins that cause mutations. One study found that APOBEC proteins, which are involved in immune activities, cause over 70% of mutations in cancers like cervical, bladder, and lung tumors. The mutations may be influenced by environmental factors. The second study was the first to perform full genome sequencing on people with autism and found genetic risk factors for autism that could lead to improved treatments and allow for earlier diagnosis through genetic screening.
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This document discusses oxidative stress and its relationship to diseases like diabetes and cancer. It proposes studying whether oxidative radicals that attack red blood cells cause variations in electrical potential. The study of cell membranes and the interactions between lipids and proteins is also discussed, noting their importance for cell structure and function. Methods like immunofluorescence, x-ray fluorescence, and crystallography are proposed to better understand these components and interactions at a molecular level.
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The document summarizes three scientific research seminars presented by students:
1. Lori Isom's research on inducing pluripotency in skin cells from an epileptic patient to create neurons and study what causes epileptic seizures. This could help treat Dravet syndrome.
2. Another student's research applying pluripotency to create specific tissues for organ repair by using a patient's own DNA.
3. Shelby Ellery's study looking at adding bridged nucleosides to drugs to chemically stabilize solutions, which could help reduce harmful side effects in patients.
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2. They engineered mammalian cells to incorporate an artificial chemical handle that allows a small fluorescent molecule to rapidly and stably attach to proteins of interest, enabling new types of cell biological studies.
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Ibm luisetto m,a.b.nili,a. h nili,hamid gamal a, et al. similarity between so...
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1. The document discusses recent discoveries about genetic variations between human genomes and structural variants that can disrupt genes.
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3. The continued investigation of protein coding in genes is important for developing new and effective medications, as well as methods for disease prevention and health promotion.
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5. angelica assignment 2 march 9 revised
This document contains summaries of three laboratory assignments:
1) A column chromatography laboratory that taught protein isolation techniques and showed lysozyme purification.
2) A neurobiology workshop that covered the nervous system, neurons, and included a sheep brain dissection.
3) An in-silico drug design workshop where students identified benzene clusters that could bind to and inhibit the HIV protease protein.
Metabolomics: The Next Generation of Biochemistry
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The paper proposes 9 hallmarks of aging based on accumulated cellular damage over time: 1) genomic instability, 2) telomere attrition, 3) epigenetic alterations, 4) loss of proteostasis, 5) deregulated nutrient sensing, 6) mitochondrial dysfunction, 7) cellular senescence, 8) stem cell exhaustion, and 9) altered intercellular communication. Some hallmarks like cellular senescence are beneficial at low levels but harmful in excess. The hallmarks represent primary damage, compensatory responses turned pathological over time, and integration of various damage types systemically.
Central dogma of genetic information
The documents discuss new research challenging the idea of the selfish gene, proposing that evolution is instead driven by the selfish ribosome. The ribosome contains the machinery for translating genetic information from DNA into proteins. Research suggests the ribosome was central to the origin of the first cell and its complex structure makes even minor mutations highly lethal for viruses. Studies of the central dogma of genetics and how life began are important for advancing medicine by providing new perspectives to approach genetic problems and find cures for diseases.
Anfinsen.pdf
The document discusses the time scale of evolution and some evolutionary principles. It outlines the major eras in geological time stretching back over 4 billion years. While the earliest fossils date to the Cambrian period 540 million years ago, life likely emerged in the Pre-Cambrian era. The document presents diagrams showing the hypothesized relationships and variations in abundance of the major phyla of life over this time period. It aims to provide context for discussing the biochemistry and genetics of different species and estimating rates of genetic modification between species.
Proteins' Structure and Function: Clinical and investigation advances.
Broschure made for a Molecular Biology class at Universidad Pontificia Bolivariana's Medicine School.
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The document summarizes several studies on DNA replication and the molecular machinery involved. It describes how researchers used tools like molecular biology and biochemistry to slow down and "freeze" the replication process at intermediate steps. This allowed them to observe conformational changes in proteins like the origin recognition complex (ORC) as it recruits the helicase enzyme to unwind DNA. Cryo-electron microscopy was then used to capture 3D structures of the protein components and reveal how they interact during replication initiation. The studies provide new insights into this critical cellular process.
Protein structure and function
Mariana Patiño Restrepo
Molecular Biology
Universidad Pontifica Bolivariana, Medellín-Colombia
2015
Plegable Biología Molecular
The document discusses several topics related to proteins and medical research. It describes the structure of proteins, the protein RAF-1 which is essential for blood vessel formation, the process of angiogenesis, and how this relates to tumor development. It also discusses the protein PGC-1 alpha and the hormone irisin which are linked to exercise and health benefits, and how isolating these proteins could lead to new treatment methods for diseases like obesity and cancer. Finally, it talks about how recent medical advances and research using body proteins/molecules could help promote healthier lifestyles and reduce disease rates.
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This document summarizes recent research on the role of autophagy in Alzheimer's disease (AD) pathogenesis and its potential as a therapeutic target. Autophagy is a cellular degradation pathway important for neuronal homeostasis that declines with age, a major risk factor for AD. Evidence suggests autophagic dysfunction may contribute to AD pathology through impaired clearance of amyloid-β protein aggregates and tau. Modulating autophagy may therefore help treat AD. However, the pathway may be less "druggable" at late disease stages. The authors propose early-stage intervention trials to target autophagy as a potential AD therapy.
Mollecular switches
This document discusses recent advances in molecular switches and protein labeling. It describes how molecular switches oscillate between 'on' and 'off' states regulated by effectors, such as the protein SRP54 interacting with SRP RNA. The document also discusses how chemists developed an improved method of labeling proteins using a 21st amino acid with a chemical handle that reacts faster and yields more labeled proteins than current methods. Understanding these molecular functions and improvements in protein labeling could help researchers better understand protein roles in cells and discover treatments for diseases.
Ibm luisetto m,a.b.nili,a. h nili,hamid gamal a, et al. similarity between so...
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1. The document discusses recent discoveries about genetic variations between human genomes and structural variants that can disrupt genes.
2. It describes how identifying these exact genetic sequences of variations can help researchers narrow their searches for disease-causing mutations and develop new treatment possibilities.
3. The continued investigation of protein coding in genes is important for developing new and effective medications, as well as methods for disease prevention and health promotion.
Biology, medicine tool
Biology and medicine have a close relationship where biology serves as an important tool for medicine. Studies on cellular metabolism and the role of proteins in cell division have helped further our understanding of diseases from the beginning. Research on a single protein that controls genetic networks essential for sperm development found that without this protein, sperm development is abnormal. A separate study on mitochondrial diseases found they are becoming more frequent and costly to healthcare systems, highlighting the need for improved prevention and treatment strategies. Advances in understanding basic biological mechanisms allow for more effective diagnosis and management of illnesses.
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Investigation about proteins brochure
- 1. INVESTIGATION ABOUT PROTEINS: BROCHURE JUAN CAMILO LOPERA RESTREPO 000175272 UNIVERSIDAD PONTIFICIA BOLIVARIANA FACULTAD DE MEDICINA TERCER SEMESTRE COLOMBIA MEDELLIN 2011
- 20. “ The future has many names: for the weak it is unattainable, for the fearful, the unknown. For the brave, the opportunity.” THANKS